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Online campaign, Times story boost efforts to save Muscat-based toddler with ailment
March 20, 2014 , 7 : 16 am GST
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An online campaign is under way in Oman, the UAE and India to save the life of this seven-month-old Indian boy. Photo - Supplied
Online campaign and a news story published by Times of Oman (
Online support gains momentum for toddler with rare ailment
) is helping the efforts being made to save the life of a Muscat-based seven-month-old Indian boy suffering from a unique genetic disorder.
Times of Oman news story
is helping us a lot in coordinating the efforts to save the baby's life. We are getting umpteen numbers of phone calls extending help. We are now confident that we will be able to save the baby's life," Saraswathy Manoj, who is coordinating the online campaign in Oman, told the Times of Oman.
A marathon online campaign is under way in Oman, the UAE and India to find 25ml of matching blood to save the life of seven-month-old Sijoy who is suffering from a genetic disorder called Severe Combined Immuno Deficiency (SCID), also known as 'bubble boy' disease.
According to medics, the baby can be saved if they get 25ml of blood from someone who is a match for the baby. The most common treatment for SCID is bone marrow transplantation, which has been successful using either a matched related or unrelated donor, or a half-matched donor, who could be either parent.
Sijoy, son of an engineer working in Muscat who hails from the south Indian state of Kerala, had earlier undergone treatment in Oman and is now in India, where he is currently under treatment.
"Blood matching camps in India will be held today (Thursday). We have found ways to hold a camp in the UAE. Here in Oman, a senior medic and a linguistic wing are looking out for options to organise the camp," Saraswathy added.
"Holding a camp also requires a huge amount of money. We are now discussing how to sort out that issue," Saraswathy added.
SCID is the most severe form of primary immunodeficiency and there are now at least nine different known genes in which mutations lead to a form of SCID.
Its victims are extremely vulnerable to infectious diseases. The SCID is the result of an immune system so highly compromised that it is considered almost absent.
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